What is the effect of alcohol (ethanol) on cellular respiration?

The deleterious effects of ethanol on a variety of tissues may result largely from altered ion permeabilities and transport. Clinically relevant ethanol concentrations in blood increase the sodium permeability of the plasma membrane and depress active sodium transport by suppressing Na, K-ATPase activity. As a result, intracellular sodium concentration increases. The total tissue content of calcium increases. Important transport mechanisms deranged by ethanol probably include those regulating calcium-sodium and hydrogen-sodium exchange at the plasma membrane and calcium uptake by the sarcoplasmic reticulum. A modest decline in magnesium content of muscle occurs after chronic exposure to ethanol. This also has been associated with accumulation of calcium. After days to weeks of sustained ethanol intake, sodium pump activity, active sodium transport and tissue oxygen consumption increase. The cell membrane potential, initially lowered by alcohol, increases to supraphysiological levels.

photosynthesis+6CO2 + 6H2O + capacity ® C6H12O6 + 6O2 a million. Carbon dioxide contained in the air is presented in by ability of the stomates (those little holes contained in the leaf). 2. Water comes into the leaf from the roots. 3. image voltaic strikes the floor of the leaf and its capacity is trapped by ability of the chlorophyll. 4. The capacity enables replace area of the water to hydrogen (HYE-druh-jun) and oxygen (OX-uh-jun). 5. The hydrogen joins with the carbon dioxide to make nutrients for the plant. 6. Oxygen gasoline comes out of the leaf by ability of the stomates. cellular respiratory=C6H12O6 + 6O2 ® 6CO2 + 6H2O + capacity Glycolysis Krebs Cycle Electron delivery Chain